Mulholland, P. ., Stepanyan, T. ., Self, R. ., Hensley, A. ., Harris, B. ., Kowalski, A. ., … Prendergast, M. . Corticosterone and dexamethasone potentiate cytotoxicity associated with oxygen-glucose deprivation in organotypic cerebellar slice cultures. Neuroscience, 136(1), 259-67. https://doi.org/10.1016/j.neuroscience.2005.07.043 (Original work published 2005)

Prendergast, M. ., Rogers, D. ., Mulholland, P. ., Littleton, J. ., LH, W. . , Jr, Self, R. ., & Nath, A. . (2002). Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl-D-aspartate receptor. Brain Research, 954(2), 300-7. https://doi.org/10.1016/s0006-8993(02)03360-7 (Original work published 2002)

Mulholland, P. ., Harris, B. ., Wilkins, L. ., Self, R. ., Blanchard, J. ., Holley, R. ., … Prendergast, M. . (1969). Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol (Fayetteville, N.Y.), 31(1-2), 1-10. https://doi.org/10.1016/j.alcohol.2003.09.001 (Original work published 1969)

Self, R. ., Mulholland, P. ., Nath, A. ., Harris, B. ., & Prendergast, M. . (2004). The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-D-aspartate receptor polyamine-sensitive site. Brain Research, 995(1), 39-45. https://doi.org/10.1016/j.brainres.2003.09.052 (Original work published 2004)

Mulholland, P. ., Self, R. ., Harris, B. ., Littleton, J. ., & Prendergast, M. . (-)-nicotine ameliorates corticosterone’s potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity. Neuroscience, 125(3), 671-82. https://doi.org/10.1016/j.neuroscience.2004.02.007 (Original work published 2004)

Mulholland, P. ., Self, R. ., Harris, B. ., Littleton, J. ., & Prendergast, M. . (2004). Choline exposure reduces potentiation of N-methyl-D-aspartate toxicity by corticosterone in the developing hippocampus. Brain Research. Developmental Brain Research, 153(2), 203-11. https://doi.org/10.1016/j.devbrainres.2004.08.008 (Original work published 2004)

Self, R. ., Mulholland, P. ., Harris, B. ., Nath, A. ., & Prendergast, M. . (2004). Cytotoxic effects of exposure to the human immunodeficiency virus type 1 protein Tat in the hippocampus are enhanced by prior ethanol treatment. Alcoholism, Clinical and Experimental Research, 28(12), 1916-24. https://doi.org/10.1097/01.alc.0000148108.93782.05

Mulholland, P. ., Self, R. ., Harris, B. ., Little, H. ., Littleton, J. ., & Prendergast, M. . (2005). Corticosterone increases damage and cytosolic calcium accumulation associated with ethanol withdrawal in rat hippocampal slice cultures. Alcoholism, Clinical and Experimental Research, 29(5), 871-81. https://doi.org/10.1097/01.alc.0000163509.27577.da

Mulholland, P. ., Self, R. ., Stepanyan, T. ., Little, H. ., Littleton, J. ., & Prendergast, M. . Thiamine deficiency in the pathogenesis of chronic ethanol-associated cerebellar damage in vitro. Neuroscience, 135(4), 1129-39. https://doi.org/10.1016/j.neuroscience.2005.06.077 (Original work published 2005)

Butler, T. ., Smith, K. ., Self, R. ., Braden, B. ., & Prendergast, M. . (2011). Neurodegenerative effects of recombinant HIV-1 Tat(1-86) are associated with inhibition of microtubule formation and oxidative stress-related reductions in microtubule-associated protein-2(a,b). Neurochemical Research, 36(5), 819-28. https://doi.org/10.1007/s11064-011-0409-2